Life-threatening anoxic spells caused by tracheal compression after repair of esophageal atresia: Correction by surgery☆
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Cited by (101)
Current concepts in tracheobronchomalacia: diagnosis and treatment
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2020, Pediatric Gastrointestinal and Liver Disease, Sixth EditionTracheobronchomalacia, Tracheobronchial Compression, and Tracheobronchial Malformations: Diagnostic and Treatment Strategies
2020, Seminars in Thoracic and Cardiovascular Surgery: Pediatric Cardiac Surgery AnnualCitation Excerpt :Surgical options include pexy procedures (ie, anterior aortopexy, anterior and/or posterior tracheopexy, anterior and/or posterior bronchopexy, posterior descending aortopexy), tracheal resection and end-to-end anastomosis or slide tracheoplasty, and placement of external splints and/or internal stents, either absorbable or permanent. Until recently, anterior aortopexy has been the mainstay of surgical treatment for intrathoracic TBM.32–35 During anterior aortopexy, the thymus is often removed or displaced to create space in the superior mediastinum, and the aorta and, frequently, innominate artery, pulmonary arteries, and/or pericardium is pulled anteriorly and sutured to the posterior surface of the sternum in an attempt to relieve airway compression (Fig. 2B).
External stenting: A reliable technique to relieve airway obstruction in small children
2017, Journal of Thoracic and Cardiovascular SurgeryComplications in neonatal surgery
2016, Seminars in Pediatric SurgeryPediatric tracheomalacia
2016, Seminars in Pediatric SurgeryCitation Excerpt :In patients with TM, the abnormal collapsibility of the trachea accentuates the physiological airway narrowing that occurs during expiration, and in severe cases clinically obvious obstruction of the airway may result when greater intrathoracic pressure increases occur, as during forced expiration or coughing.7 In the extreme case, complete obstruction to the outflow of air may lead to a fatal outcome.8 In addition, the lack of normal tracheal stiffness in TM allows tracheal collapse from compression by adjacent thoracic structures, mainly the aortic arch and innominate artery anteriorly and the esophagus posteriorly.9
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Presented before the 7th Annual Meeting of the American Pediatric Surgical Association. Boca Raton, Fla., April 29–May 1, 1976.