EditorialBeta blockers prevent cardiac death following a myocardial infarction: So why are so many infarct survivors discharged without beta blockers?
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Cited by (41)
Beta-blockers reduced the risk of cardiac rupture in patients with acute myocardial infarction: A meta-analysis of randomized control trials
2017, International Journal of CardiologyCitation Excerpt :There are neither means of predicting or monitoring CR, nor an effective therapy. Beta-blockers have long been a mainstay for the treatment of acute MI [5]. Clinical studies have shown that early beta-blockers treatment in acute MI patients reduce total mortality and sudden cardiac death [6,7].
Preoperative Evaluation
2015, Murray and Nadel's Textbook of Respiratory Medicine: Volume 1,2, Sixth EditionEarly intravenous beta-blockers in patients with acute coronary syndrome-A meta-analysis of randomized trials
2013, International Journal of CardiologyCitation Excerpt :Beta-blockers have long been a mainstay for the treatment of acute coronary syndromes (ACS) [1].
Effect of bisoprolol on respiratory function and exercise capacity in chronic obstructive pulmonary disease
2012, American Journal of CardiologyThe role of β-blockers as a cornerstone of cardiovascular therapy
2005, American Journal of HypertensionImpact of intravenous Beta-Blockade before primary angioplasty on survival in patients undergoing mechanical reperfusion therapy for acute myocardial infarction
2004, Journal of the American College of CardiologyCitation Excerpt :Although detailed explanations as to why beta-blockers were withheld were not collected, it is unlikely that strict contraindications were present in most patients. It is well known that beta-blockers are frequently under-prescribed in the peri-infarct period (24,25), possibly reflecting physicians' concerns about their safety in the AMI setting and misconceptions regarding absolute versus relative contraindications (26). In the present study, intravenous beta-blocker administration was safe, being associated only with a slight increase in the incidence of transient peri-procedural heart failure, without increases in adverse events such as bradyarrhythmias or severe hypotension.